Fig. 6From: The role of REST and HDAC2 in epigenetic dysregulation of Nav1.5 and nNav1.5 expression in breast cancerTSA caused dose- and time-dependent growth suppression in MCF-7 cells. MCF-7 cells were treated with TSA at concentration of 10–10,000 ng/ml for 24–72 h and cell growth was measured using MTT assay. The cell growth of untreated control cells was considered as 100%. Data were collected from n = 3 independent experiments, presented as mean ± SEMBack to article page