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Fig. 1 | Cancer Cell International

Fig. 1

From: HOXC6 gene silencing inhibits epithelial-mesenchymal transition and cell viability through the TGF-β/smad signaling pathway in cervical carcinoma cells

Fig. 1

Overexpressed HOXC6 contributes to the occurrence of CC. a Positive expression of HOXC6 protein in CC tissues and paracancerous tissues (× 200); scale bar = 50 μm; b positive rate of HOXC6 protein between in CC and paracancerous tissues; c relative expression of HOXC6, TGF-β1, TGF-βRII, smad4, and smad7 in CC tissues (n = 44) and paracancerous tissues (n = 13) detected by RT-qPCR; d linear interaction of HOXC6 to TGF-β1, TGF-β R II, smad4, and smad7 analyzed using Pearson correlation analysis; n = 72; *p < 0.05 compared with paracancerou tissues. CC cervical carcinoma, HOXC6 homeobox C6, RT-qPCR reverse transcription quantitative polymerase chain reaction, TGF transforming growth factor, smad solvated metal atom dispersion

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