Skip to main content
Fig. 1 | Cancer Cell International

Fig. 1

From: BMX-ARHGAP fusion protein maintains the tumorigenicity of gastric cancer stem cells by activating the JAK/STAT3 signaling pathway

Fig. 1

High BMX-ARHGAP expression is observed in GC tissues and cells. a BMX-ARHGAP expression in GC tissues and adjacent normal tissues determined by RT-qPCR; b the BMX-ARHGAP expression in GC cell lines (SNU-5, MNK-45, AGS and SGC7901) and normal gastric epithelial cell line (GES-1) determined by RT-qPCR; c, d the BMX-ARHGAP protein expression in GC cell lines (SNU-5, MNK-45, AGS and SGC7901) and normal gastric epithelial cell line (GES-1) measured by western blot assay. *p < 0.05 vs. the adjacent normal tissues or GES-1 cells. Enumeration data in a were expressed as mean ± standard deviation and compared with paired t test; measurement data in b, d were expressed as mean ± standard deviation and analyzed by one-way analysis of variance; each experiment was repeated three times. BMX bone marrow kinase on chromosome X, ARHGAP RhoA GTPase activating protein, RT-qPCR reverse transcription quantitative polymerase chain reaction, GC gastric cancer

Back to article page

Cancer Cell International

ISSN: 1475-2867

Contact us