Skip to main content
Fig. 7 | Cancer Cell International

Fig. 7

From: β-Asarone increases doxorubicin sensitivity by suppressing NF-κB signaling and abolishes doxorubicin-induced enrichment of stem-like population by destabilizing Bmi1

Fig. 7

Schematic diagram showing the action of β-Asarone in the induction of apoptosis and suppression of stem-like properties in lymphoma cells. β-Asarone functioned as an inhibitor of the stem-like population in lymphoma cells by destabilizing Bmi1 via a proteasome-mediated mechanism and promoting intracellular doxorubicin uptake. β-Asarone also functioned as a sensitizer to induce synergistic effects on proliferation and apoptosis when combined with doxorubicin treatment by blocking NF-κB signaling activation

Back to article page