Fig. 2
From: Mechanistic study of lncRNA UCA1 promoting growth and cisplatin resistance in lung adenocarcinoma
UCA1 contributed to the proliferative capacity of LUAD cells. A, B Lentivirus-mediated UCA1 overexpression and knockdown in A549 cells and A549/DDP cells were examined by RT-qPCR. C, D Growth curves of cells were evaluated by CCK-8 assays after overexpressing and knocking down UCA1 in A549 cells and A549/DDP cells. E, F Protein expression levels of PCNA in A549 cells and A549/DDP cells were determined by Western blot analysis, which indicated that UCA1 promoted LUAD cell proliferation. *P < 0.05, **P < 0.01, ***P < 0.001