Fig. 4
From: SYT7 acts as an oncogene and a potential therapeutic target and was regulated by ΔNp63α in HNSCC
Knockdown of SYT7 suppressed the proliferation of HN6 and CAL-27 cells and ΔNP63α-SYT7 double knockdown could partially rescue this phenotype. A, B Detection of cell proliferation of SYT7-knockdown HN6 cells by Celigo cell count. C, D Detection of cell proliferation of ΔNP63α SYT7 double knockdown HN6 cells by Celigo cell count. E, F Detection of cell proliferation of SYT7-knockdown CAL-27 cells by Celigo cell count. G, H Detection of cell proliferation of ΔNP63α SYT7 double knockdown CAL-27 cells by Celigo cell count (n = 3). Bars show the mean ± SD. ***Represents p < 0.001. Scale bar = 100 μm