Fig. 1
From: SYT7 acts as an oncogene and a potential therapeutic target and was regulated by ΔNp63α in HNSCC
Overexpression of ΔNP63α inhibited growth and migration of HN6 and CAL-27 cells. A Detection of cell proliferation of HN6 cells by Celigo cell count. B Detection of cell proliferation of CAL-27 cells by Celigo cell count. C Wound-healing assay of HN6 cells. D Wound-healing assay of CAL-27 cells (n = 3). Bars show the mean ± SD. ***Represents p < 0.001. Scale bar = 100 μm