Open Access

Erratum to: Functional disruption of macrophage migration inhibitory factor (MIF) suppresses proliferation of human h460 lung cancer cells by caspase-dependent apoptosis

Cancer Cell International201313:84

https://doi.org/10.1186/1475-2867-13-84

Received: 20 August 2013

Accepted: 20 August 2013

Published: 20 August 2013

The original article was published in Cancer Cell International 2013 13:28

Correction

After publication of the original article [1] it came to the authors attention that an incomplete version of Figure three (Figure 1 here) was published with the article. The complete figure and new figure legend are presented in this correction article.
Figure 1

siRNA-mediated knockdown of MIF expression in H460 cells detected by Western blot. (A) is a western blot of MIF expression in H460 cells treated with NC and two MIF siRNAs for 48 hours. In MIF siRNA-transfected H460 cells, we observed an approximately two (siRNA 1) to five (siRNA2) fold weaker signal of MIF protein expression compared with the negative control (NC) group normalized to the expression of β-actin. (B) is a densitometric analysis of the western blot. This shows the relative densities of protein levels which were measured by Quantity One (Bio-Rad company) software.

Notes

Declarations

Authors’ Affiliations

(1)
Department of Pulmonary Medicine, the First Affiliated Hospital of Sun Yat-Sen University
(2)
Department of Physiopathology, Zhongshan School of Medicine, Sun Yat-Sen University

References

  1. Guo Y, Hou J, Luo Y, Wang D: Functional disruption of macrophage migration inhibitory factor (MIF) suppresses proliferation of human H460 lung cancer cells by caspase-dependent apoptosis. Cancer Cell Int. 2013, 13: 28-10.1186/1475-2867-13-28.PubMed CentralView ArticlePubMedGoogle Scholar

Copyright

© Guo et al.; licensee BioMed Central Ltd. 2013

This article is published under license to BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/2.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

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