Skip to main content

Retraction Note to: Excessive mitochondrial fragmentation triggered by erlotinib promotes pancreatic cancer PANC‑1 cell apoptosis via activating the mROS‑HtrA2/Omi pathways

Retraction to: Cancer Cell Int (2018) 18:165 https://doi.org/10.1186/s12935-018-0665-1

The authors have retracted this article [1] because Figure 4a has been duplicated from Figure 5d in a previously published article [2]. In addition, the article contains sections that substantially overlap with the following articles (amongst others) [3,4,5]. The data reported in this article are therefore unreliable. Author Hong Ke stated on behalf of all co-authors that they agree to this retraction.

References

  1. 1.

    Wan J, Cui J, Wang L, et al. Excessive mitochondrial fragmentation triggered by erlotinib promotes pancreatic cancer PANC-1 cell apoptosis via activating the mROS-HtrA2/Omi pathways. Cancer Cell Int. 2018;18:165. https://doi.org/10.1186/s12935-018-0665-1.

    CAS  Article  PubMed  PubMed Central  Google Scholar 

  2. 2.

    Liu J, Yan W, Zhao X, et al. Sirt3 attenuates post-infarction cardiac injury via inhibiting mitochondrial fission and normalization of AMPK–Drp1 pathways. Cell Signal. 2019;53:1. https://doi.org/10.1016/j.cellsig.2018.09.009.

    CAS  Article  PubMed  Google Scholar 

  3. 3.

    He M, Xiang Z, Xu L, et al. Lipopolysaccharide induces human olfactory ensheathing glial apoptosis by promoting mitochondrial dysfunction and activating the JNK–Bnip3–Bax pathway. Cell Stress Chaperones. 2019;24:91. https://doi.org/10.1007/s12192-018-0945-7.

    CAS  Article  PubMed  Google Scholar 

  4. 4.

    Xie Y, Lv Y, Zhang Y, et al. LATS2 promotes apoptosis in non-small cell lung cancer A549 cells via triggering Mff-dependent mitochondrial fission and activating the JNK signaling pathway. Biomed Pharmacother. 2019;109:679. https://doi.org/10.1016/j.biopha.2018.10.097.

    CAS  Article  PubMed  Google Scholar 

  5. 5.

    Chen Z, Wang C, Yu N, et al. INF2 regulates oxidative stress-induced apoptosis in epidermal HaCaT cells by modulating the HIF1 signaling pathway. Biomed Pharmacother. 2019;111:151. https://doi.org/10.1016/j.biopha.2018.12.046.

    CAS  Article  PubMed  Google Scholar 

Download references

Author information

Affiliations

Authors

Corresponding author

Correspondence to Hong Ke.

Additional information

Publisher's Note

Springer Nature remains neutral with regard to jurisdictional claims in published maps and institutional affiliations.

Rights and permissions

Open Access This article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

Reprints and Permissions

About this article

Verify currency and authenticity via CrossMark

Cite this article

Wan, J., Cui, J., Wang, L. et al. Retraction Note to: Excessive mitochondrial fragmentation triggered by erlotinib promotes pancreatic cancer PANC‑1 cell apoptosis via activating the mROS‑HtrA2/Omi pathways. Cancer Cell Int 19, 286 (2019). https://doi.org/10.1186/s12935-019-1019-3

Download citation